I was just conserned because I already have insomina, and my doctor knows this.  My guess is he is trying to use the adderall to keep me up and moving during the day so I can sleep better at night (which I've already texercise daily so I don't quiet follow his logic). 

 

He's a man of few words so he rarely tells me exactly what he is thinking/planning.  I know this isn't a good quality in a doctor but I've been to 4 others who all sucked and, quiet frankly, I'm tired of this "doctor shopping."  Either way, I'll be back in his office in 7 days to discuss effectiveness, progress, and the like.

 

On the plus side, he also prescirbed me some benzo's to take at night if I am still feeling "buzzed"/awake from the Adderall.

 

 

 

P.S. Addy XR doesn't come in a generic (not till 2018 or some time close to that) and this **edited** costs almost $7 a pill, it better friggin' work miracles!

 

I'm going to make the (risky) assumption that he's a doctor, and thus went to medical school, and therefore: he has a much much larger knowledge in this field than I ever will. 

 

Missed this line before.  I typically do try and ask pharmacists questions and sometime I get good answers but, more often than not, I get halfassed answers.  The one time I asked if this medication was going to have life threatening reactions with cheese and other stuff containing thryosine, he proceeded to take the piece of paper that they give you with every presciption and read it to me....Needless to say my question was never answers.

 

I seem to always have the worst luck with doctors/pharmacists/veterinarians

 

My father baught me a PDR, however I forgot to bring it to school with me.   If I had it then I could find these answers myself.

 

 

 

 

 

 

|D 0 U |3 L 3 P 0 5 T

 

My neurobiology class has treated me well so far, lets see what else I can recall to answer your question:

 

Cocaine/meth are CNS stimulate that FLOODS the synapse with dopamine/serotonin/norepinephrin, which inturn almost constantly stimulate the next door neighbor neuron (cocaine goes so far as to go backwards into the nerve cell and open up more channels, so that more neurotransmitters are released even without a stimulus).

 

Benzo's bind to the GABA-recceptor on CNS neurons, and increase it's affinity for the neurotransmitter gamma butrytic acid (GABA) (sp?).  This makes more GABA bind to the GABA-receptor, when this happens it hyperpolaries the cell (via potassium influx).  All in all, this makes the nerve slow its function down a LOT.

 

So with all this dopamine/norepi/serotonin swimming around and stimulating the nerves, the benzo's go around (and stick around for a long time) and slowly "turn the neurons off."  And thus, you lose you "come down" much quicker.

 

If you find my answer unacceptable then I suggest you Google it. I'm sure you'll find it out there somewhere.  It's a common practice among meth/coke users.

 

 

 

 

It's a very long story, but it will suffice to say that I had the displeasure of spending a huge portion of my time with a veterinarian whom I absolutely CAN NOT stand.  Every minute around him made me want to pull one of those "murder-suicides" that I see on TV. 

 

 

Similar mechanism, obviously a lot more intense because of some varations in how it works.